PeterLuffman
Level 5 Valued Member
I understand there is growing evidence and research that documents the negative effects of HIIT training?
Does anyone have any links to actual studies?
Does anyone have any links to actual studies?
Thanks for the reply.
More specifically, I'm referring to the material for Pavel's Strong Endurance seminar. I was under the impression he goes into this topic. Just wondering if there's any reading material available before I attend next month.
@PeterLuffman
It goes into some of the “why”. There is also some good stuff on AGT on this blog, from our own @miked:
Why do antiglycolytic training? - Part 1
Acidosis overrides oxygen deprivation to maintain mitochondrial function and cell survival
BDNF important for age-effected cognitive function and memory loss. Any aerobic work helps this, HIIT helps a little bit more.High-intensity interval training evokes larger serum BDNF levels compared with intense continuous exercise.
Training prescriptions to induce mitochondrial biogenesis in untrained individuals therefore seem to be relatively straight forward: if exercise duration is relatively long, there is no point in exercising harder than 65-70% of VO2max. However... if time is a limiting factor, HIIT protocols, especially SIT (sprint interval), are the most time efficient training strategies to promote mitochondrial biogenesis.
...HIIT is also a time efficient strategy to reduce muscle glycogen. A single 30 s all-out sprint can reduce muscle glycogen by as much as 25% (Barnett et al., 2004)and 8x60 s sprints can reduce glycogen by more than 75% (Gollnick et al., 1974). Further, HIIT seemed to reduce glycogen to a greater extent in fast type II fibers than in type I (Thomson et al., 1979).
It has also been shown that HIIT increases mitochondrial biogenesis(SDH-activity) more in type-II fibers than in type-I (Henriksson and Reitman, 1976). The efficacy of HIIT training to induce mitochondrial biogenesis might therefore be a consequence of its ability to effectively deplete some muscle fibers of their glycogen.
...an increase in [Pi] can cause fatigue through 2 of the 3 mechanisms by which pH was once believed to do so...Furthermore, there is a lack of association between changes in pH and MVC (maximal voluntary contraction) throughout fatiguing exercise and in recovery in humans. The recent evidence regarding the role of pH in muscle fatigue may help to dispel previously held misconceptions about the development of muscle fatigue.