Abdul-Rasheed
Level 6 Valued Member
Thank you @North Coast Miller . Much appreciated.
Compared to someone who's ketogenic, if you shut off the fats and gave them only carbs to survive they might need medical attention before their insulin response normalized.
"Fat dependent" might be a better description than "fat adapted". The non-keto individual is more metabolically flexible.
The non-keto individual is more metabolically flexible.
...the lipid sources are a slower reaction to recharge ATP compared to glucose.
"Carbohydrate Dependent" means they they are dependent of glucose. These individual's system are inefficient at utilizing body fat. That due to the fact that they are Metabolically Inflexible.
Kenny Croxdale
That is not supported by any science, at least regarding physically active folks. People with healthy mitochodrial function and some aerobic conditioning have no problem processing fats for ATP.
Keto adapted folk have an advantage over non keto when it comes to larger % of fat in the diet, but many if not all have impaired glucose response when in this state, overall less flexible - my earlier statement stands.
Depends on your sport. For powerlifting, it works well.I've never met one who was on a low carb high fat diet, let alone been ketogenic,
It always comes down to physics in the end..."The remarkable feature of physical laws is that they apply everywhere, whether or not you choose to believe in them. After the laws of physics, everything else is opinion."
2. Replacement of saturated fat with naturally occurring unsaturated fats provides health benefits for the general population.
You need to expand you research on this. The premise is build on a house of cards.
ATP Restoration Recovery
Glucose vs Lipid Restoration Time?
I take it that you don't know, which is fine. I have spoken with a few Exercise Physiologist that use vague terms and no definitive answer.
This also makes no sense. I might claim glucose supplies as much ATP per unit...
.The answer "it depends" is the correct one.
You need to expand you research on this. The premise is build on a house of cards.
The above results are not new. They were emphasized by Odin (3), Malmros (4), Stenstrom (5), Staub (6), Kageura (7), Greenwald, Gross, and Samet (8) and others. It was believed that during a period of a low carbohydrate, high fat diet, the need for insulin was diminished, with a resulting decrease in its production. Then, upon administration of a large quantity of carbohydrate to a subject subsisting on such a diet, the carbohydrate mechanism is heavily taxed. The production of insulin cannot keep up with the demand, the result being a hyperglycemia and often a glycosuria as well. The extensive and excellent work of Malmros (4) also supports the above view. He worked with normal human beings who had glucose tolerance tests after general diets as well as after high fat, low protein, and low carbohydrate mixtures. The duration of such diets varied from 1 to 23 days.
In every case a lowering of the tolerance to carbohydrate was noted irrespective of the duration of the preceding diet. Greenwald, Gross, and Samet (8) were of the same opinion. They believed that a diet consisting chiefly of protein does not lower the tolerance for glucose as much as a diet consisting principally of fat.
If then a low carbohydrate, low protein, and high fat diet lowers the tolerance to carbohydrate in normal human beings, is it reasonable to assume that a high carbohydrate diet will raise the tolerance?
This too has been demonstrated.
Other research data that I have indicates ketones may yield up to 4 time more ATP than glucose.